electron microscope em 10 cr (Carl Zeiss)
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Carl Zeiss
electron microscope em 10 cr
Electron Microscope Em 10 Cr, supplied by Carl Zeiss, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/electron+microscope+em+10+cr/902+electron+microscope/pm39936986-78-9-10
Average 90 stars, based on 1 article reviews
Electron Microscope Em 10 Cr, supplied by Carl Zeiss, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/electron+microscope+em+10+cr/902+electron+microscope/pm39936986-78-9-10
Average 90 stars, based on 1 article reviews
electron microscope em 10 cr - by Bioz Stars,
2026-11
90/100 stars
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other:Article Title: A differentiated porcine bronchial epithelial cell culture model for studying human adenovirus tropism and virulence. Article Snippet: The species specificity of human adenoviruses (HAdV) almost precludes studying virulence and tropism in animal models, e.g. rodent models, or derived tissue and cell culture models.. However, replication of HAdV type 5 (HAdV-C5) has been shown after intravenous injection in swine.. In order to study adenovirus replication in airway tissue propagation of bronchial epithelial cells from porcine lungs was established. Article Title: Magnetic resonance imaging monitoring dual-labeled stem cells for treatment of mouse nerve injury. Article Snippet: Background aims.. Adipose-derived stem cells (ADSCs) have shown great promise in the regenerative repair of injured peripheral nerves.. Magnetic resonance imaging (MRI) has provided attractive advantages in tracking superparamagnetic iron oxide nanoparticle (SPION)-labeled cells and evaluating their fate after cell transplantation. Article Title: Farnesoid X Receptor Agonism Protects against Diabetic Tubulopathy: Potential Add-On Therapy for Diabetic Nephropathy Article Snippet: Ultrathin sections were cut with a Reichert Ultracut S ultrami- crotome (Leica Microsystems, Wetzlar, Germany), contrasted with uranyl acetate and lead citrate, and were viewed with an EM 10 Article Title: Defective podocyte insulin signalling through p85-XBP1 promotes ATF6-dependent maladaptive ER-stress response in diabetic nephropathy. Article Snippet: Ultrathin sections were cut with a Reichert Ultracut S ultramicrotome (Leica Microsystems, Wetzlar, Germany), contrasted with uranyl acetate and lead citrate and were viewed with an EM 10 Microscopy:Article Title: Distinct domains in Ndc1 mediate its interaction with the Nup84 complex and the nuclear membrane. Article Snippet: Nuclear pore complexes (NPCs) are embedded in the nuclear envelope and built from ∼30 different nucleoporins (Nups) in multiple copies, few are integral membrane proteins.. One of these transmembrane nucleoporins, Ndc1, is thought to function in NPC assembly at the fused inner and outer nuclear membranes.. Here, we show a direct interaction of Ndc1’s transmembrane domain with Nup120 and Nup133, members of the pore membrane coating Y-complex. Article Title: Synaptic Activity Protects Neurons Against Calcium-Mediated Oxidation and Contraction of Mitochondria During Excitotoxicity. Article Snippet: Th is pa pe r h as b ee n pe er -re vi ew ed a nd a cc ep te d fo r p ub lic at io n, b ut h as y et to u nd er go co py ed iti ng a nd p ro of co rr ec tio n. T he fi na l p ub lis he d ve rs io n m ay d iff er fr om th is pr oo f. FORUM ORIGINAL RESEARCH COMMUNICATION Title: Synaptic activity protects neurons against calcium-mediated oxidation and contraction of mitochondria during excitotoxicity Abbreviated Title for running head: Neuronal activity reduces mitochondrial oxidation Authors: Constanze Depp, Carlos Bas-Orth, Lisa Schroeder, Andrea Hellwig, Hilmar Bading Both authors contributed equally to this work.. Department of Neurobiology, Interdisciplinary Center for Neurosciences, Heidelberg University, Im Neuenheimer Feld 364, 69120 Heidelberg, Germany To whom correspondence should be addressed: Prof. Hilmar Bading, Department of Neurobiology, Interdisciplinary Center for Neurosciences, Heidelberg University, Im Neuenheimer Feld 364, 69120 Heidelberg, Germany; Telephone: +49 6221 548218; FAX: +49 6221 546700; E-mail: Bading@nbio.uni-heidelberg.de Article Title: N-methyl-d-aspartate Receptor-mediated Preconditioning Mitigates Excitotoxicity in Human Induced Pluripotent Stem Cell-derived Brain Organoids. Article Snippet: Studies in rodent models of acute and chronic neurodegenerative disorders have uncovered that glutamate-induced excitotoxic cell death is mediated primarily by extrasynaptic N-methyl-D-aspartate receptors (NMDARs).. Rodent neurons can also build up in an activity-dependent manner a protective shield against excitotoxicity.. This form of acquired neuroprotection is induced by preconditioning with low doses of NMDA or by activation of synaptic NMDARs triggered by bursts of action potentials. Article Title: Pharmacological Targeting of the NMDAR/TRPM4 Death Signaling Complex with a TwinF Interface Inhibitor Prevents Excitotoxicity-Associated Dendritic Blebbing and Organelle Damage. Article Snippet: Ultrathin sections were cut with a Reichert Ultracut S ultramicrotome (Leica Microsystems, Wetzlar, Germany) and contrasted with uranyl acetate and lead citrate. .. Specimens were visualized using an |